...RAD51 foci formation at damage sites. Using a tissue microarray of chemoresistant EAC patients, we observed that ISG15 i...
...RAD51 foci were found in DDX41 KO and DDX41-R525H expressing cells, indicating a defect in the transition from end resec...
...RAD51) and nonhomologous end joining (via KU70/80). This irreparable DNA damage resulted in G2/M phase arrest and activa...
...RAD51 paralogs, suggesting a shared HR program. Functionally, transient knockdown of FOXM1 or MYC reduced BRCA1/RAD51 tr...
...RAD51. Importantly, BRCA1 knockdown attenuated SERBP1-driven cisplatin resistance both in vitro and in vivo, establishin...
...RAD51 were abnormally activated in the adjacent BMSCs. However, after knockdown of IFT88 (SiIFT88), the protein expressi...
...Rad51, pCtIP), EdU/CCK-8 (proliferation), TUNEL/flow cytometry (apoptosis), comet assay (DNA damage), CldU/IdU (replicat...
...RAD51/BRCA2-mediated stabilization, PCNA modifications, PARP1- and CTC1-STN1-TEN1 (CST)-dependent fill-in pathways, and ...
...rad51, etc.) and collaborates with AURKA for cell cycle progression. In vivo experiments demonstrated that Rab5b reduced...
...RAD51, RPA2 and cGAS, showed mutually exclusive localization with importin α in MN, indicating that MN comprise function...
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